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    Thread: using anabolic steroids without hpta shutdown

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      using anabolic steroids without hpta shutdown

      RESEARCH SHOWS THAT NOT ALL STEROIDS CAUSE SHUTDOWN!
      *NO MORE POST-CYCLE CRASH!
      *You can now USE CERTAIN STEROIDS DURING PCT! *(Pre-PCT)
      *You can now formulate a cycle that will NOT CAUSE SHUTDOWN
      Some steroids only REDUCE TESTOSTERONE PRODUCTION(to varying degrees), whereas other steroids will SHUTDOWN the HPTA resulting in a complete cessation of androgen production.
      NOT ALL ANDROGENS CAUSE SHUTDOWN
      "Shutdown", is defined by a COMPLETE inhibition of the Pituitary/Testes, resulting in a TOTAL cessation of endogenous androgen production.
      SOME androgens will only SUPPRESS endogenous androgen production, resulting in a DECREASED testosterone level, but not a complete shutdown. (Turinabol, Anavar, Halotestin, Wistrol, Equipoise, Dianabol, Masteron, Primobolan)
      Very Androgenic/Progestenic/Estrogenic steroids(Trenbolone, Nandrolone, Anadrol, Testosterone) cause a COMPLETE shutdown of endogenous hormone production.
      The distinction between SUPRESSION and SHUTDOWN is utterly important, as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle, and a quicker, easier recovery!
      The Following steroids will NOT SHUTDOWN THE HPTA:
      Turinabol, Anavar, Proviron, Halotestin, Wistrol, Equipoise, Dianabol, Masteron, Primobolan, Clostebol, and 4-ADiol.
      Pre-PCT: PRE-PCT allows the HPTA to begin LH/FSH output, while still receiving additional anabolic support. This is the peroid of time where we utilize a NON-inhibitory steroid while the endogenous testosterone level begins to recover. This occurs PRIOR TO FULL PCT, so that by the time we begin full PCT the HPTA has already began recovering.
      Active RECOVERY: The HPTA BEGINS to restore endogenous testosterone production once it detects the body's androgen level beginning to decline(end of cycle).
      Therefore, HPTA CAN BEGIN TO RECOVER WHILE STILL IN AN ANABOLIC STATE!
      The following drugs can be used during Active Recovery:
      Anavar/Proviron= 40mgs/25mgs
      Anavar/Masteron= 40mgs/300mgs
      Primobolan/Masteron= 300mgs/300mgs
      Turinabol/Proviron= 40mgs/25mgs
      Turinabol/Masteron= 40mgs/300mgs
      Winstrol/Masteron= 50mgs/300mgs
      Dianabol/Proviron= 15mgs/25mgs
      Dianabol/Masteron= 15mgs/300mgs

      Examples...
      In a SHORT CYCLE:
      Weeks 1-4: Testosterone Propionate, 100mgs ED
      Weeks 1-4: Dianabol, 50mgs ED
      Weeks 1-4: NPP, 400mgs
      Weeks 4-8: PRE-PCT(ACTIVE RECOVERY)
      Weeks 8-?: POST CYCLE THERAPY

      A Standard Cycle:
      Weeks 1-6: Dianabol, 30mgs ED
      Weeks 1-10: Testosterone Enanthate, 500mgs
      Weeks 8-12: Winstrol, 100mgs ED
      Weeks 12-16: PRE-PCT(ACTIVE RECOVERY) **
      Weeks 16-26: **POST CYCLE THERAPY

      DO NOT end your cycle ABRUPTLY! Don't just END your cycle cold-turkey! If you are SHUTDOWN, full restoration can take weeks and even MONTHS. Therefore, one should REMAIN ON minimally-inhibitive STEROIDS(HPTA) in an attempt to MAINTAIN the gains they made while ON CYCLE, while STILL BEGINNING TO RECOVER TESTOSTERONE PRODUCTION. On top of that, one still continues to progess from the mild additional anabolic support.
      NOT only does it mean that you can run a COMPLETE CYCLE with NO SHUTDOWN whatsoever(as long as the right compounds, dosages, and durations are used), it also means that if you ARE SHUTDOWN from your cycle, you do NOT HAVE TO COME RIGHT OFF CYCLE! Actually, it is BETTER TO STAY ON CYCLE WHILE YOUR ENDOGENOUS TESTOSTERONE LEVEL BEGINS TO INCREASE!
      You may also run a cycle that COMPLETELY AVOIDS SHUTDOWN:
      Weeks 1-6: Dianabol, 40mgs ED
      Weeks 1-10: Anavar, 50mgs ED
      Weeks 1-10: Masteron, 100mgs EOD

      Or
      Weeks 1-6: Dianabol, 40mgs ED
      Weeks 1-10: Primobolan, 500mgs
      Weeks 6-14: Turinabol, 60mgs ED

      And Many many more! There are tons of NON-inhibitory cycles that you can devise using my my list above for your guideline. Your days of HPTA suffering are over!
      By understanding WHICH steroids cause SHUTDOWN and which steroids do NOT, we can formulate a perfect EXTENDED CYCLE.
      The Hypothalamus has Androgen, Estrogen, and Progesterone receptors.
      Each and EVERY anabolic steroid affects these receptors DIFFERENTLY.
      Some steroids affect ALL receptors, while some only affect ONE type of receptor, while others have very little effect on ANY of these receptors.
      UNDERSTANDING WHICH steroids affect which receptors, and to WHAT DEGREE, will FULLY enable the steroid user to COMPLETELY and systematically AVOID HPTA SHUTDOWN!
      By understanding WHICH steroids cause SHUTDOWN and which steroids do NOT, we can formulate a perfect EXTENDED CYCLE.
      Steroids that cause an OVERSATURATION(too many receptors activated) of these various hormone receptors, WILL CAUSE SHUTDOWN.
      Steroids that DO NOT CAUSE an OVERSATURATION of ANY of these various hormone receptors, will NOT cause SHUTDOWN!
      The Following drugs either DIRECTLY or INDIRECTLY activate ESTROGEN receptors, to varying degrees:
      Testosterone
      Methandrostenolone
      Mathandriol
      Oxymetholone
      Nandrolone
      Boldenone

      The Following drugs either DIRECTLY or INDIRECTLY activate PROGESTERONE receptors, to varying degrees:
      Nandrolone
      Trenbolone
      Oxymetholone

      The Following drugs activate Androgen receptors, to varying degrees:
      Testosterone
      Methandrostenolone
      Mathandriol
      Oxymetholone
      Nandrolone
      Boldenone
      Trenbolone
      Halotestin
      Oxandrolone
      Stanzolol
      Chlorodehydromethltestosterone
      Methyltestosterone
      Methenolone...
      (ALL AAS*)

      As we can see, the steroids that cause HPTA SHUTDOWN either OVERSATURATE ONE SPECIFIC receptor, or they activate too many TOTAL receptors(Androgen/Estrogen/Progesterone)
      For instance, Trenbolone causes HPTA SHUTDOWN because it OVERSATURATES BOTH, the ANDROGEN and the PROGESTERONE receptors.
      Testosterone causes SHUTDOWN because it converts to ESTROGEN and DHT, therefore, it oversaturates the Androgen/Estrogen receptors.
      As we can ALSO SEE, the steroids that DO NOT cause SHUTDOWN of the HPTA, do NOT oversaturate ANY of the different hormone receptors, and thus, do NOT cause SHUTDOWN.
      Methenolone(Primobolan) does not possess ANY Estrogenic or Progestational ACTIVITY WHATSOEVER. It does, by virtue of being an anabolic steroid, posses a SMALL Androgenic component. Because it lacks ANY ESTROGENIC/PROGESTATIONAL component, and it lacks a strong Androgenic component, it WILL NOT CAUSE SHUTDOWN!
      Oxandrolone(Anavar) posseses NO Estrogenic/Progestational component either. AND, it also lacks a strong androgenic component. Thus, Anavar will NOT cause shutdown.
      By understanding WHICH steroids cause SHUTDOWN and which steroids do NOT, we can formulate a perfect EXTENDED CYCLE.
      *It must also be noted, that ANY steroid in LARGE enough DOSAGES for long enough DURATIONS, can cause SHUTDOWN of the HPTA.
      NOT ALL ANDROGENS CAUSE SHUTDOWN*
      "Shutdown", is defined by a COMPLETE inhibition of the Pituitary/Testes, resulting in a TOTAL cessation of endogenous androgen production.
      SOME androgens will only SUPPRESS endogenous androgen production, resulting in a DECREASED testosterone level, but not a complete shutdown. (Tbol, Var, Wistrol, EQ, Dianabol, masteron, proviron, halo, primo)
      Very Androgenic/Progestenic/Estrogenic steroids(Tren, Deca, Drol, Test) cause a COMPLETE shutdown of endogenous hormone production.
      The distinction between SUPRESSION and SHUTDOWN is utterly important, as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle, and a quicker, easier PCT.

      source Steroids Forum: Steroids QA/Anabolic Steroids/*Using Anabolic Steroids Without HPTA SHUTDOWN!*

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      Shure wish there were case studies cited.
      PAIN IS WEAKNESS LEAVING THE BODY
      SO DON'T BE A PUSSY


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      Agreed. No references at all to support the claims
      Quote Originally Posted by Hatebreed View Post
      Shure wish there were case studies cited.

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      yep it would .

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      Effect of Non Aromatizable Androgens on LHRH and TRH Responses in Primary Testicular Failure

      I.*M.*Spitz, E.*J.*Margalioth, Y.*Yeger, Y.*Livshin, E.*Zylber-Haran, S.*Shilo

      Center for Biomedical Research, Population Council, New York, U.S.A.,

      Departments of Obstetrics and Gynecology, Bikkur Cholim Hospital, Shaare Zedek Hospital and Hadassah University Hospital, Jerusalem, Israel


      Summary

      We have assessed the gonadotropin, TSH and PRL responses to the non aromatizable androgens, mesterolone and fluoxymestrone, in 27 patients with primary testicular failure. All patients were given a bolus of LHRH (100 ug) and TRH (200 ug) at zero time. Nine subjects received a further bolus of TRH at 30 mins. The latter were then given mesterolone 150 mg daily for 6 weeks. The remaining subjects received fluoxymesterone 5 mg daily for 4 weeks and 10 mg daily for 2 weeks. On the last day of the androgen administration, the subjects were re-challenged with LHRH and TRH according to the identical protocol.

      When compared to controls, the patients had normal circulating levels of testosterone, estradiol, PRL and thyroid hormones. However, basal LH, FSH and TSH levels, as well as gonadotropin responses to LHRH and TSH and PRL responses to TRH, were increased.

      Mesterolone administration produced no changes in steroids, thyroid hormones, gonadotropins nor PRL. There was, however, a reduction in the integrated and incremental TSH secretion after TRH. Fluoxymesterone administration was accompanied by a reduction in thyroid binding globulin (with associated decreases in T3*and increases in T3*resin uptake). The free T4*index was unaltered, which implies that thyroid function was unchanged. In addition, during fluoxymesterone administration, there was a reduction in testosterone, gonadotropins and LH response to LHRH. Basal TSH did not vary, but there was a reduction in the peak and integrated TSH response to TRH. PRL levels were unaltered during fluoixymesterone treatment. These results suggest that the increased TSH response to TRH in testicular failure may be related to a decrease in androgen action.

      I can not find the rest now but I am trying.
      Last edited by minotauros; 11-27-2017 at 07:45 AM.

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      Hey brothers whats going on.

      I dont believe this to be true. I know a lot of guys use higher doses so these small doses might seem to make sense but that is not the case. I like to micro dose for athletic purposes test prop 30mg eod and anavar 40mg ed for 4 weeks and i DO get shut down. I even experience some shrinkage towards the end of the cycle. The shutdown is minimal and i recover pretty quick. Let me add that i only cycle 1-2 times a year. Every body reacts differently but i believe if it goes in, then your body shuts down. I think its more of the level of shut down than anything else. Just my 2 cents.

      Love this place

      Peace and love

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      Honestly, I just cringe every time I see posts like this. xlr8mechanism, I am not coming at you, I am sure this is well intended but honestly it is just not so. I have seen threads like these come and go for more than a decade. I have seen, heard, and participated in the debates. I appreciate the intent but in all reality this is just not the case and is frankly dangerous "advice" to throw out there. Bottom line, EVERYTHING will shut you down to one degree or another. No way around that. PCT is a must or be prepared for TRT. Honestly, even with solid PCT, in time, TRT may still be a reality. Trust me, been there, done that. TRT for life now....

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      Quote Originally Posted by Theone189 View Post
      Hey brothers whats going on.

      I dont believe this to be true. I know a lot of guys use higher doses so these small doses might seem to make sense but that is not the case. I like to micro dose for athletic purposes test prop 30mg eod and anavar 40mg ed for 4 weeks and i DO get shut down. I even experience some shrinkage towards the end of the cycle. The shutdown is minimal and i recover pretty quick. Let me add that i only cycle 1-2 times a year. Every body reacts differently but i believe if it goes in, then your body shuts down. I think its more of the level of shut down than anything else. Just my 2 cents.

      Love this place

      Peace and love
      That testicular shrinkage (as you said) is highly individual. I was on megadoses of all kinds of roids for 6 years pretty much solid and i only lost a slight amount of size in my left testicle(about 25%). The right would have no difference what so ever.

      When i would be on gh, i wouldnt even lose that amount.

      I was off for 3 plus years...maybe 4 before i started back up. Lost all my roid weight then started putting muscle on naturally (locked up). Shit tons of calisthenics all day plus what ever i could make weights from including human weight till level drop and a,weight pile..
      I got out jacked and thought..."hmmm.. if I can out on muscle at totally off everything this long with a shitty diet and in my 40's, then u wonder what 100mg test/wk and 50mg tren /wk would do?..."
      Well i found out: i blew the fuck up!!! I looked like i was on more than a gram of shit.
      I still micro dose unless im experimenting but even then, its usually experimenting with the synergy of conpounds...
      Like my next experiment will be 100mg 1 test cyp/wk + 100mg trest dec/wk + 200 mast en/wk.
      Mast doesn't due much anabolically speaking - even less with that dose but the combined aas total is 400mg substance a week and each has something the other lacks class wise and real world use wise.
      I ALWAYS do a very high volume workout.
      And if you've seen the pix ive posted (That i get paranoid as fuck and delete) youll see i look as I talk).

      Sent from my SM-J327P using Tapatalk

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      Quote Originally Posted by babybull34 View Post
      Honestly, I just cringe every time I see posts like this. xlr8mechanism, I am not coming at you, I am sure this is well intended but honestly it is just not so. I have seen threads like these come and go for more than a decade. I have seen, heard, and participated in the debates. I appreciate the intent but in all reality this is just not the case and is frankly dangerous "advice" to throw out there. Bottom line, EVERYTHING will shut you down to one degree or another. No way around that. PCT is a must or be prepared for TRT. Honestly, even with solid PCT, in time, TRT may still be a reality. Trust me, been there, done that. TRT for life now....

      anytime something strays from the standard it always has to be questioned. i do agree i should post things that are backed up with medical trials and i will use a little more discretion in the future .but if you can see my history on my browser my posts are mild . i do some searches into stuff that is far out there . but without curiousity unless stumbling upon we will never know if there is anything better out there imagine when the concept of pct using serms was proposed it must of been Heresy and now look at what most of us do now

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      Hey Lokthai,

      Great info my brother. Got a bit hyped to see another fellow microdoser.

      I also blow up on small amounts. Its nice to hear someone with same results/issues.

      How long are your microdose cycles? From past exp, what would you recommend as an anavar alternative? I stick to test p/var, great results with pretty much no sides when microdosing. Im looking for more of the same

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