Androgenic: Pertaining to the development of male characteristics, including body hair, the genital organs and muscle mass. "Androgenic" is the adjective form of the noun "androgen," a word referring to any of the male hormones, including testosterone and androsterone.
Androgenic development -- that is, the development of male characteristics -- begins in puberty, the time when a person becomes physically capable of producing offspring. In males, this time most commonly occurs between ages 12 and 14. A deepening voice is one of the signs of androgenic activity.
Androgen is produced in males by the testes, the two globe-shaped reproductive organs below the penis, and by the adrenal glands, two small hormone-producing organs that each sit atop a kidney. Androgen is also produced in females in the adrenal glands. Overproduction of androgen can generate some male characteristics in women and exaggerate male characteristics in men. "Androgenic" is derived from the Greek words "andros" (man) and "genein" (to produce)." Related words include "androgynous" (having both male and female characteristics), "andrology" (the study of health in males), "androphobia" (fear of men) and "android" (in science fiction, a manlike robot).

Anabolism (Greek "mound" from ana = upward + ballein = "to throw") is the set of metabolic pathways that construct molecules from smaller units.[1] These reactions require energy. One way of categorizing metabolic processes, whether at the cellular, organ or organism level is as 'anabolic' or as 'catabolic', which is the opposite. Anabolism is powered by catabolism, where large molecules are broken down into smaller parts and then used up in respiration. Many anabolic processes are powered by adenosine triphosphate (ATP).[2]
Anabolic processes tend toward "building up" organs and tissues. These processes produce growth and differentiation of cells and increase in body size, a process that involves synthesis of complex molecules. Examples of anabolic processes include the growth and mineralization of bone and increases in muscle mass.
Endocrinologists have traditionally classified hormones as anabolic or catabolic, depending on which part of metabolism they stimulate. The classic anabolic hormones are the anabolic steroids, which stimulate protein synthesis and muscle growth. The balance between anabolism and catabolism is also regulated by circadian rhythms, with processes such as glucose metabolism fluctuating to match an animal's normal periods of activity throughout the day

Some examples of the anabolic effects of these hormones are increased protein synthesis from amino acids, increased appetite, increased bone remodeling and growth, and stimulation of bone marrow, which increases the production of red blood cells. Through a number of mechanisms anabolic steroids stimulate the formation of muscle cells and hence cause an increase in the size of skeletal muscles, leading to increased strength.[26][27][28]
The androgenic effects of AAS are numerous. Processes affected include pubertal growth, sebaceous gland oil production, and sexuality (especially in fetal development). Some examples of virilizing effects are growth of the clitoris in females and the penis in male children (the adult penis does not grow even when exposed to high doses of androgens), increased growth of androgen-sensitive hair (pubic, beard, chest, and limb hair), increased vocal cord size, deepening the voice, increased libido, suppression of natural sex hormones, and impaired production of sperm.[29]
The androgenic:anabolic ratio of an AAS is an important factor when determining the clinical application of these compounds. Compounds with a high ratio of androgenic to a anabolic effects are the drug of choice in androgen-replacement therapy (e.g. treating hypogonadism in males), whereas compounds with a reduced androgenic:anabolic ratio are preferred for anemia, osteoporosis, and to reverse protein loss following trauma, surgery or prolonged immobilization. Determination of androgenic:anabolic ratio is typically performed in animal studies, which has led to the marketing of some compounds claimed to have anabolic activity with weak androgenic effects. This disassociation is less marked in humans, where all anabolic steroids have significant androgenic effects.[16]
A commonly used protocol for determining the androgenic:anabolic ratio, dating back to the 1950s, uses the relative weights of ventral prostate (VP) and levator ani muscle (LA) of male rats. The VP weight is an indicator of the androgenic effect, while the LA weight is an indicator of the anabolic effect. Two or more batches of rats are castrated and given no treatment and respectively some AAS of interest. The LA/VP ratio for an AAS is calculated as the ratio of LA/VP weight gains produced by the treatment with that compound using castrated but untreated rats as baseline: (LAc,t–LAc)/(VPc,t–VPc). The LA/VP weight gain ratio from rat experiments is not unitary for testosterone (typically 0.3–0.4), but it's normalized for presentation purposes, and used as basis of comparison for other AAS, which have their androgenic:anabolic ratios scaled accordingly (as shown in the table above).[30][22] In the early 2000s this procedure was standardized and generalized throughout OECD in what is now known as the Hershberger assay.


We can make it simple for our purpose's and say one is a strength builder while the other is a better mass builder.

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