So, your go-to support is that the act of body building is more about injections even if a drug has contradictions by different administration it will always be superior by way of injection, because, pussies don't inject? Interesting..LOL
Do you know anything about Oxandrolone and it's toxicity reports, or even it's oral administration bioavailability percentile? Clearly you don't
(It's 97% with up to 9-10 hrs half life, that's damn fucking good and it's not primarily metabolized by the liver).. On paper if a drug is good orally, imagine it being in an injection forum, hmm, this is the mentality of most AAS users, when in fact people fail to recognize that some drugs (there's tons of oral meds to speak of) simply don't need IM/Sub-q routes for many mechanism of actions that take place..Oral administration will facilitate,transport and give the proper uptake that Oxan needs, because of its position it has no barriers or interactions unlike MANY other oral AAS drugs that go through a series of breakdowns that dictate and freeing its availability.. If anything, placing Oxan in a OILY injection form will place it in a "depot" position, slower time release due to the carrier vehicle oil along with many other factors, thus a reason medical practice sees no purpose of value for this route if they did, it would have been used by now.. Now mainly because unlike other compounds that may undergo extensive first-pass metabolism by way of the liver that could hinder them inactive, Oxan doesn't! It's great and very effective just as it is LOL..
Bear in mind it's protected by a 17-carbon, thus it's why its used clinically and therapeutically in oral application.. Imagine that! Same reason some drugs come in orals, injection oil or aqueous, and even creams and gels for that matter, they are structured and positioned for an intend reason..LOLOL
Placing such compounds in a oily suspension/carrier as your route of exposure the absorption will slower for the lipid soluble parenting hormone that is now at the mercy of the ester/half-life of the carrier oil, and solutions.. Now, if we use a carrier or solvent/solution that has a very short half-life (rapid metabolism or absorption such as aqueous) now we ultimately eliminate the process in which the drug is released making it's plasma fast, but VERY short! You can't have STRONGER and more bioavailability, with LONGER HALF LIFE while being superior all in one, and alone just changes the whole dynamics or chemistry all due to wishful thinking.. There's a trade off with everything and this will also come at the expensive of the rapid deployment and activation of enzymes at the injection site and the drugs reaction there after, there will be a chute of responses..A spike in BP would be something to greatly consider..
There's ton of research out there, look it up, I won't dig it up for you, because that would be to much of a "convenience" for you
I love my oils and my orals, but I found that cutting corners aren't always what they appear to be.. If something is more convenient for someone, I won't tell them what they can and can't do, all power to them.. But if someone say's "This works this way because I said so",
welp, let's talk it..
To all readers, this has already became a topic, let's talk about it... Leave "anecdotal evidence" aside , because that means nothings.. "I snort my dbol and it works"... See how easy that way?
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