Anti-Estrogen Usage to Protect Your Physique and Your Physiology
Nolvadex VS Clomid?
Nolvadex
Nolvadex is one of the most well known anti-estrogens. It is also one of the oldest and most popular medications for breast cancer victims, which is the text book clinical use for this drug. Nolvadex is actually a weak estrogen and will bind to the estrogen receptor. The receptor is then occupied and when the normal and stronger estrogen floats by the receptor, it has no where to “park”. This is what we have defined as the classic second line inhibitor. Second line, because we are allowing estrogen to be produced, we are just blocking it at the receptor. For male usage, nolvadex is a good drug of choice to use for ant-bloating and to control gyno if you are not particularly susceptible to these estrogenic side effects. If you are not and just need minor control, 20mg per day is probably enough to keep the majority of water off and your nips from being sensitive. If you are susceptible, you may need a stronger ant-estrogen, or may need to combine nolvadex with another anti-estrogenic drug, or may need a much higher dosage of nolvadex (probably closer to 40mg/day).
There is some literature out there that states nolvadex having better leutinizing (testosterone stimulating) properties than clomid. This is with respect to post cycle recovery and jump starting your natural testosterone. Personally, amongst all my friends and colleagues, I have not found this to be true. With respect to post cycle recovery of natural testosterone production clomid is definitely more effective.
Clomid
As we’ve stated above, clomid is more effective at restoring natural testosterone production. Everything you read about these two drugs seems to offer the same info. I will afford you with something different. For anti-estrogen protection, nolvadex is probably a little stronger. Its chemical structure makes for a slightly more potent anti-estrogen.
I have yet to see any quality literature which describes the affinity for either drug at the estrogen receptor. Clomid after all, is also a weak estrogen. Another term used to classify clomid and nolvadex is “competitive inhibitor”. They are so called due to the fact that they are weak estrogens and actually bind to the receptor. By doing this they are competing for the “parking spot” in the receptor, thereby occupying the space when the truer, stronger estrogen comes along. Its like being at the local mall and having someone pull around the isle, cut you off and take your spot. In a sense they just “competed” with you for your spot and “inhibited” you from taking it. Got it? Good. See this stuff is simple, my fellow friends and athletes.
The literature also fails to describe exactly why clomid works more efficiently as a leutinizer. Even in the medical literature, the drug is primarily used for fertility purposes for both males and females. This means clomid has value in stimulating the pituitary in both men and women. Clomid is used on a five day course in order to stimulate ovulation in the female in order to enable fertilization. There is not a fertility doctor in the world that doesn’t know what clomid is. It is a drug that has always been in their toolbox. Part of the reason is, it works. The literature is less extensive for fertility cases in the male but know that it is indeed used for this purpose. Now, as much as I am all for underground self taught knowledge, we have to hand it to the medical professionals for specific cases. For example, if nolvadex were a better leutinizer, it would be the drug of choice in fertility clinics for stimulating ovulation in the female and for aiding fertility and motility of sperm in the male. Simple fact is that this is not the case. Save your self time and frustration by using a medically time tested drug for your post cycle recovery of testosterone. For recovery of your natural test, use clomid.

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